Outline and evaluate the assumptions made by two models of abnormality

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Outline and evaluate the assumptions made by two models

about the causes of abnormality

One model of abnormality is known as the medical or sometimes the biological model.  This model assumes that all behaviour is rooted in underlying physiological processes in the body, and therefore that abnormal behaviour is some sort of malfunction in the body, or perhaps due to genetic factors.  Following on from this, the model suggests that it is possible to ‘cure’ abnormality, and it is done so by fixing the malfunction in the body or returning it to normal levels of functioning.  This is why it has the medical name - the model assumes abnormality to be a mental illness, in the same way that physical illness is considered a medical problem that can be treated medically.  The biological model is widely respected and influential, owing to its heavy scientific basis.

The main assumption of the biological model of abnormality is that abnormality is caused by physical factors such as genes, biochemical substances, infection and brain injury (possibly since birth).  There is also evidence to suggest that the role of neurotransmitters can affect abnormality, for example serotonin levels have been linked to depression, and dopamine with schizophrenia.  What the biological model fails to acknowledge however is precisely why these levels might change.   Whilst the cause of physical illness is often possible to trace, it is not the case with psychological disorders, even though the biological model might try to justify medically.

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The biological model also promotes the idea of cause and effect.  Again using the case of neurotransmitter levels associated with psychological disorders, when a patient is treated with medicine designed to bring these levels back to normal, improvements seen go some way to proving that the neurotransmitter levels were responsible.  However, such medications will only treat what could arguably be just a symptom - as mentioned, the biological model fails to consider what causes the change in the first place.  Further to this, it may be that the neurotransmitter levels are themselves a symptom of the psychological disorder, rather ...

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