Explanations of Eating Disorders.

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Explanations of Eating Disorders

Biological Explanations

These fall into two categories – genetic inheritance or an imbalance of neurotransmitters in the brain.

Genetic Explanation;

  • Science unable to identify genes for specific behaviour such as those associated with eating disorders
  • Research is based on trends in families
  • American Psychiatric Association reports from statistics that there is increased risk of eating disorders amongst first-degree biological relatives of those diagnosed.
  • However relatives usually share the same environment [nature vs. nurture]
  • Twin studies provide more reliable evidence. In comparing monozygotic (MZ) twins with dizygotic (DZ) twins, a significantly higher concordance rate was found among MZ twins compared with DZ twins.
  • Twin research into anorexia by Holland et al.  found a 55% concordance rate for MZ twins compared to only 7% for DZ twins
  • Kendler et al. found similar results for bulimia nervosa, with 23% for MZ twins and 8.7% for DZ twins.
  • Even though the results from these studies show very high concordance rates, in Holland’s study it still leaves 45% of MZ twins who are discordant.
  • Hsu suggested that the genetic element might relate to personality traits which make the person more susceptible to stressful events. Which could manifest in the form of an eating disorder.
  • In many cases of anorexia and bulimia, there is family history of mood or personality disorders.
  • Wade et al studied both genetic and environmental risk factors in 325 female twins, and found significant environmental influence in shaping women’s attitudes towards weight, shape, eating and food … but little evidence of a genetic component.

Biochemical Explanation;

  • Eating disorders may be associated with a biochemical imbalance. Research of the brain has focused on the region known as the “hypothalamus.”
  • Animals have been found to stop eating or even starve themselves to death when this part of the brain is damaged.
  • According to Keesey and Corbett, the lateral hypothalamus and the ventromedial hypothalamus work alongside each other to provide a ‘weight thermostat,’ sending messages about being hungry/full up.
  • A malfunction in this part of the hypothalamus offers a possible explanation for eating disorders although there is as yet no conclusive evidence
  • Amenorrhoea can occur before weight loss, suggesting a primary disorder of low endocrine levels – which is also associated with hypothalamus dysfunction.
  • More recent research has focused on certain hormonal chemicals (norepinephrine, dopamine and low levels of serotonin…associated with binge eating)
  • Walsh et al. have found that most effective drug treatment for bulimia is serotonin-active antidepressant medication.
  • Jimmerson et al. conducted clinical tests comparing serotonin functions in patients with bulimia compared to healthy controls. They found considerable differences and concluded that impaired serotonergic responsiveness may contribute to the abnormal eating patters in people with bulimia.
  • The problem with biochemical research is that is difficult to differentiate between the cause and the effect i.e. starvation may eventually cause an imbalance in biochemical functioning.
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Behavioural Explanations

  • Classical Conditioning incorporates the layperson’s view, suggesting that slimming becomes a habit, through stimulus-response mechanisms. They learn to associate being slim with feeling good about themselves
  • Operant Conditioning comes into play as admiration from others further reinforces the dieting behaviour. Refusing to eat my also provide an additional reward in gaining attention from parents, and starving oneself can even be rewarding as an effective way of punishment.
  • Cross-cultural studies seem to support the behavioural explanation. Anorexia and bulimia are more prevalent in industrialised societies like Europe, USA, Canada, and Australia etc. In these societies ...

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