Burns Case Study. The pathophysiology involves both local and systemic responses which Klein et al. (2009) explains affects skin or other tissue and is dependent on the cause of the injury and extent.

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Running head: BURNS

BURNS

SPC BELL, ALEXANDRA

BURNS

Christenson and Kockrow (2011) states that a burn can be defined as an injury to the tissue which results from thermal, chemical, radiation, or electrical. Thermal burns are caused by a flame, hot fluids or gases, friction, or exposure to extremely cold objects. Flame burns are most often associated with smoke or inhalation injury.  Chemical burns occur when there is contact with a caustic substance. This substance can be acidic or alkaline. The degree of injury is relative to the type and content as well as the concentration and temperature of the injuring agent. Electrical burn is when a current travels through the body along the pathway of least resistance. This pathway often includes nerves, as they offer the least resistance, while the greater resistance is through bones. The resistance from bones can generate heat. The amount of injury is dependent on the type and voltage of current. Electrical burns often have underlying injury which is sometimes more severe than the observable injury. Doenges, Moorhouse, and Murr (2010) explains that radiation burns result from an exposure to an ionizing radiation, most commonly is an overexposure to ultraviolet rays from the sun, sunlamp, tanning booths, or high exposure to x-rays.

Doenges et al. (2010) suggests that there are several risk factors predispose a person to burns including substance abuse, careless smoking, cultural practices, overcrowded living conditions, lack of parental supervision of children, insufficient safety precautions and violence, including child abuse and neglect.

The pathophysiology involves both local and systemic responses which Klein et al. (2009) explains affects skin or other tissue and is dependent on the cause of the injury and extent.  In local response Klein et al. (2009) explains that there are three stages. The first stage is coagulation which occurs at the point of maximum damage, causing irreversible tissue loss due to coagulation of the constituent proteins.  The stasis stage is the area characterized by a decrease in tissue perfusion which is viable unless prolonged hypotension, infection, or edema, occur. Hyperemia is the outermost area, which has increased tissue perfusion, this tissue will often recover.  Immediately after a burn injury cytokines and other inflammatory mediators are released at the site of the injury. This can result in an increased capillary permeability and leads to a shift of intravascular proteins and fluids into the interstitial space.

According to Doenges et al. (2010) burns are classified by four degrees. The first degree or superficial partial thickness burn only affects the epidermis, the skin is often warm and dry, and wounds typically appear bright pink to red with minimal edema and fine blisters. Second degree or moderate partial thickness burns, include the epidermis and dermis. Second degree wounds appear red to pink with moderate edema and blisters that may be intact or draining. Second degree burns can also be deep partial thickness. These burns extend into the deep dermis and are dryer than moderate partial thickness burns. They appear pale pink to pale ivory, with moderate edema and blisters. Third degree or full thickness burns include all layers of skin and subcutaneous fat. They may also involve the muscle, nerves, and blood supply. These wounds have a dry, leathery texture and appearance varies from white to cherry red to brown or black, with blistering uncommon. Often there is an absence of pain in the center, but the edges of the burn wound may have heightened sensation. Fourth degree or full thickness subdermal burns involve all skin layers as well as muscle, organ tissue, and bone.

Doenges et al. (2010) states that in electrical burns patient’s signs and symptoms observed may include mixed areas of numbness, tingling, burning pain, changes in orientation, changes in vision, decreased visual acuity, decreased deep tendon reflexes, seizure activity, and paralysis. Doenges et al. (2010) goes on to explain that in almost all burns patients will present with tachycardia, tissue edema formation, decreased urinary output, decreased or absent bowel sounds, nausea, vomiting, facial mask, changes in blood pressure, pulse, and increased respiratory rate. Breath sounds in an inhalation injury may have crackles, stridor, or wheezing. Doenges et al. (2010) says that hoarseness, wheezy cough, carbonaceous particles on face or in sputum, drooling or inability to swallow oral secretions, cherry colored face, and cyanosis, altered mental status are all signs and symptoms which may point to carbon monoxide poisoning.

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There are several diagnostic procedures that can be used to assess the extent of damage when treating a patient with a burn injury. Klein et al. (2009) explains that the first diagnostic information collected is the total body surface area or TBSA, which is calculated with the rule of nines. In the rule of nines the major anatomical areas of the body can be divided into percentages, in adults, nine percent for the head and neck, nine percent for each upper extremity, eighteen percent to each of the anterior and posterior portions of the trunk, eighteen percent to each lower ...

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