Section 1

St Swithins Hospital

Physiotherapy Documentation

Database

Patient name: Mr. Jack Jones                                      D.O.B: 4/4/50

Address: The Lodge, Hollyoaks Estate                          Ward: HDU

PC: Perforated gastric ulcer

HPC: Admitted via ambulance

  • Pt found semi-conscious on floor at home by wife
  • Complaining of increased epigastric pain over night after meal out
  • Vomiting AM- frank blood
  • Hx of epigastric pain over last few months

PMH: Hypertension

         High cholesterol

         COPD (recently diagnosed)

DH: Lipostat

       Captopril

       Ventolin inhaler 

        

SH: Married, 2 grown up children

       Smoker, 15-20 per day for 40 years

       Alcohol, 10 units per week

       Sales manager                                                                

                                                                    Word:97

Physiotherapy Problem List

                                                                                                   

                                                                                                 Word:188

Day 1

S: Pt returned from theatre at 6pm last night, following repair of perforated gastric ulcer and partial gastrectomy. Significant blood loss- 5 units transfused. CVS unstable throughout. Central line, arterial line, epidural and urinary catheter in-situ. N/S report rattly sounds on cough. Pt reluctant to do anything.

On HDU

O: Obs. Pt in bed. SV with venturi mask in situ.

     SaO2: 94% n 50% O2

     RR: 19 tachypneoa      

     ABG’S from 7.30am: Ph- 7.44      

                                    PCO2- 4.5 kPa  

                                    HCO3- 22

                                    PO2- 10.2 kPa hypoxaemic

      BP:105/75

      HR: 105 tachycardic

      Temp: 37.6 slight pyretic

      CVP: 3

      Palp:  basal expansion L

      Ausc: Breath Sound (BS) throughout but  L base.  No added sounds.

      CXR shows: Collapse L base,  costophrenic angles, heart & mediastinum L shift

      Fluids: IV2L, oral 0, Urine output 12L  (10ml/hr)

     

 Investigations/Results: Urea 6.2mmol/L

                                   Creatine 114mmol/L                                Word:325                            

                                   Hb 9

                                   Platlets 120*10(9)/L

                                   WCC 6

Medication:

  • Noradrenaline
  • Paracetamol
  • Bupivicaine
  • Omeprazole

A:  

  • lung volume (2° atelectasis)
  • V/Q mismatch
  • Unstable CVS
  • Acute renal failure.
  • Unproductive cough

Mr. Jones is suffering from decreased lung volume in base of his left lung, secondary to atelectasis (deflated alveoli). His recent surgery, history of chest infections with the latest diagnosis of COPD, along with his smoking history are all factors that increase the risk of developing atelectasis and other post pulmonary complications (Brooks-Brunn 1995). Decreased expansion on left base is the result of the deflated alveoli. Atelectasis is particularly common after chest or abdominal surgery because the effects positioning or pain of deep breathing (Brooks-Brunn 1995). Decreased breath sounds left base also suggest atelectasis, as air is unable to reach the base due to collapse of the alveoli. Chest x-ray A confirms this showing collapse left lower lung.                                                               Word:477                

Mr Jones shows signs of hypoxemia, with PO2 of 10.2kpa and SaO2 94%.  Ventilation is commonly affected by the reduced lung volume. In turn, gas exchange properties between oxygen and blood are diminished, as the oxygen required is hindered. This creates V/Q mismatch resulting in hypoxemia (Roussos & Koutsoukou 2003).

Evidence suggests superficial secretions due to reported ‘rattly sounds’ by nursing staff. They are concerned as patient is finding it difficult to cough, which is to be suspected, as pain from the abdominal incision will reduce voluntary cough. However, suppression of cough could lead to the accumulation of secretions increasing risk of infection (Dilworth & Poundsford 1991).

The high concentrations of urea and creatinine create concern for the development of acute renal failure. His fluid output is low, showing olguria (Short & Cumming 1999). However, this is common after surgery.

Mr. Jones shows signs of unstable CVS. He is tachycardic with HR of 105 and low CVP of 3. This is treated by the medical staff with the use of appropriate medication.

P:

  • Position change
  • Breathing exercises – ACBT
  • Teach supported cough

                                                                                                 Word:661                                        

Liaise with M/S about need for humidification as adequate hydration will reduce possible sputum retention and further atelectasis (Jackson 1995)

Liaise with nursing staff re-CVS, prior to positioning. Position patient in right high side lying, making sure he is fully supported. Turning from supine to side lying can clear atelectasis, enhancing gas exchange. This will increase V/Q mismatch and help clear secretions (Stiller 2000). Caution must be taken when handling patient with regard to the wound incision. Alterations to high sitting would be advisable to reduce risk of developing pressure sores (Thomas 2006).

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ACBT would be indicated with all three components used. BC will help reduce his breathlessness. TEE would be carried out as able, to increase expansion. Holds would be avoided due to breathlessness. FET would be included, as huffing to low lung volumes helps to clear the secretions from peripheral  (Bhowmik et al, 2008). Cycle would include: BC-BC-BC-TEE-BC-BC-BC-FET-BC-BC-BC (twice)

Constant monitoring of hemodynamic status is vital, to detect deleterious side effects of treatment (Stiller et al, 2004).

Supported cough would be taught, to relieve pain and fear when coughing. It will coincide with the increase in airflow following the new ...

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