The astonishing feature of these studies was that when dogs were exposed to inescapable shock they adopted behaviour patterns that were passive in nature, even when they were given continued shocks they remained inactive.

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More than thirty-five years ago Seligman and his colleagues discovered and reported on two experiments which were to begin an ever-growing field of research, and to fuel continuing debates (Overmeir & Seligman, 1967; Seligman & Maier, 1967). The astonishing feature of these studies was that when dogs were exposed to inescapable shock they adopted behaviour patterns that were passive in nature, even when they were given continued shocks they remained inactive. This phenomenon was labelled “learned helplessness” and instigated a theory around the findings. The theory postulated that only inescapable events produced giving up, because the identical pattern of shock, if it was under the animal’s control, did not produce giving up. The design used for these types of experiments was known as the “yoked triadic design” which involved three groups of subjects. A contingent group: These subjects are allowed to escape, or avoid a stimulus. A non-contingent group: These subjects are exposed to a stimulus of the same duration by a yoking procedure. And, a no treatment group: These subjects are not exposed to any response outcome contingencies (Hejka, 1994). What this experimental design assumed was that animals in the non-contingent group have the cognitive ability to learn that their actions are futile and therefore will no longer try to escape, instead they will become passive. Therefore, the original learned helplessness theory hypothesised that instead of learning occurring only when a response produces a reward or punishment, much to the behaviourists’ acknowledgement; it can occur independent of whether or not a reward or punishment has been given.

Maier and Seligman (1976) and Seligman (1975) developed the original learned helplessness hypothesis, which proposed that exposing organisms to uncontrollable outcomes (outcomes that are independent of responding) would produce a motivational deficit, a cognitive deficit and an emotional deficit. Seligman (1975) also postulated that human depression shares similar symptoms to that of learned helplessness and that the two conditions may be instigated by the same factors, that non-aversive outcomes can cause learned helplessness, and learned helplessness can be applied to all organisms not just dogs and humans.

The early experiments investigating the phenomenon of learned helplessness were confined to animal subjects, but it was not long before it was tested in the human context (Hiroto & Seligman, 1975). Indeed, a number of early studies set out to test that symptoms of depression paralleled the original theory of learned helplessness, which involved depressed subjects and their ability to solve anagram patterns and to turn off inescapable noise (Klein, Fencil-Morse & Seligman, 1976; Miller & Seligman, 1975; Klein & Seligman, 1976). It must be noted however, that not all early studies demonstrated the effect of learned helplessness in other animal species as Seligman had suggested it would (Seligman, 1975). Indeed, a study conducted by Beatty (1979, cited in Hejka, 1994) failed to show any decrements in test task performance of rats following inescapable shock, while others found an improvement in test task performance (Anisman, & Waller 1971, cited in Hejka, 1994).

The original theory sparked much debate and critiques to the original theory of learned helplessness were intensifying. The behaviourists’ headed the list believing that the animals’ had learned to remain passive, which would thus give a reward of the shock being turned off. Others believed that subjects differed in their sensitivity to shock. While some would be able to sit there and take the shock, others were said to have a low tolerance and would therefore try to escape, and the sensitivity subjects had to the shock fluctuated over time. As evident as Seligman thought the early studies were in supporting the phenomenon of learned helplessness resulting in the same symptoms seen in naturally occurring depression, the theory still failed to account for ‘boundary conditions’, such as two out of three people becoming helpless but one resisting and no matter what happened to them to make them helpless, they would not give up (Peterson, Maier, & Seligman, 1993).

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In the reformulated theory (Abramson, Seligman & Teasdale, 1978) cognition was refined in the form of attribution which postulated that human behaviour is controlled not just by the schedule of reinforcement in the environment but by an internal mental state, the explanations people make for why the environment has scheduled their reinforcements this way. More specifically the reformulated theory focussed on the following three dimensions of attributions for negative life events: internal-external, stable-unstable and global-specific (Abramson et al., 1978). Internal attributions explain causes of negative events in self-referent terms, whereas external attributions assign causes to factors outside the self. ...

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