Critically evaluate research into eating disorders - Anorexia Nervosa and Bulimia Nervosa.

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Jack Waters

Critically evaluate research into eating disorders

Anorexia Nervosa and Bulimia Nervosa

Anorexia Nervosa and Bulimia Nervosa are the two main types of eating disorders that have been found, mainly in the western world.

The term Anorexia Nervosa means Loss of appetite, but sufferers of Anorexia Nervosa do not actually have a loss of appetite, in fact most of the people with Anorexia have normal appetites and will most likely be very hungry, but they will still behave as if they have lost their appetite. The A.P.A (American Psychiatric Association) (1994) states that Anorexia occurs in 0.5-1.0 % of females in late adolescence or early adulthood.

   The main characteristic of Anorexia Nervosa is the constant obsession of bodyweight and sufferers will usually be under the minimum expected bodyweight but will still be afraid of putting on weight or gaining body fat. Their food intake is usually restricted to 600-700 calories per day.

   Bulimia Nervosa sufferers are very different they tend to induce themselves in secret binge eating, but then rid their bodies of the food usually by self induced vomiting, the over use of laxatives or sometimes by excessive exercising. Unlike Anorexics, Bulimics are usually within the expected bodyweight, but like Anorexia, the sufferers have a distorted body image of themselves and those of people they model themselves upon (supermodels, pop singers, etc).

   The Biological model tends to treat abnormality’s, such as eating disorders, as physical illness’s and it is believed that treatment of these disorders should be dealt with in a physical way, such as drug treatment (chemotherapy). One explanation of why these eating disorders occur is that there has been a dysfunction of the neurotransmitters in the brain.

    Keesey and Corbett (1983) did some research into the lateral hypothalamus (LH) and the Ventromedial Hypothalamus (VMH). They discovered that the LH and the VMH work alongside each other to create a weight thermostat, The LH produces the feeling of hunger and the VMH depresses the feeling of hunger. If the persons bodyweight falls below the set point on the weight thermostat the LH is activated and the person will feel hungry and will want to eat something, but if the persons bodyweight rises above a set point on the thermostat then the VMH will be activated and the person should stop feeling hungry, and supposedly stop eating. A malfunction in this system is a possible cause for eating disorders (the lack of LH and an excess activation of VMH), although there is no conclusive evidence to support this yet.

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   Another Biological explanation of eating disorders is that they are genetically inherited. The A.P.A (1994) stated that, from diagnostic statistics, there is an increased risk of eating disorders amongst first-degree Biological relatives (siblings, parents and children). Research done by Holland et al (1984), into monozygotic twins (MZ) and dizygotic twins (DZ), MZ twins have exactly the same genes whereas DZ twins need be no more alike then any normal siblings. The nature of this research was to see if MZ twins have a higher concordance (meaning that they both have the same disorder) rate then DZ twins in ...

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